Figure 2
Measurement of cardiac functions and myeloperoxidase activities. Increase in the peak rate of intraventricular pressure increase (+dP/dtmax: A), decrease in the peak rate of intraventricular pressure (−dP/dtmax: B) in rats 72 hours, and cardiac myeloperoxidase activity (MPO: C) in rats 24 hours after ischemia-reperfusion injury. Rats were treated with: endothelial progenitor cells (EPCs) (150 μL, 5 × 106 cells per rat); EPCs transfected with Tβ4 short hairpin RNA (shRNA) (150 μL, 5 × 106 cells per rat); EPCs transfected with scrambled (SC) shRNA (150 μL, 5 × 106 cells per rat); or 6 mg Tβ4. Control rats were untreated. *P < 0.05 compared with the control group; †P < 0.05 compared with the Tβ4 group; ‡P < 0.05 compared with the EPC group; §P < 0.05 compared with the EPCs + SC shRNA group. Data are presented as mean ± standard deviation (n = 8 per group).